Proceedings of the Third International Symposium
Herausgegeben von Schettler, G.; Weizel, A.
Softcover reprint of the original 1st ed. 1974. 2011. xxxviii, 1036 S. XXXV, 1034 pp. 349 figs., 222 ta
Verlag/Jahr: SPRINGER, BERLIN 2011
ISBN: 3-642-65956-X (364265956X)
Neue ISBN: 978-3-642-65956-0 (9783642659560)
´I. The Arterial Wall in the Pathogenesis of Atherosclerosis.- Workshop: The Composition of Early Lesions.- The Lesion.- Metabolism of the Arterial Wall.- Biophysical Factors in Vascular Structure and Caliber.- Contraction of Endothelial Cells as a Key Mechanism in Atherogenesis and Treatment of Atherosclerosis with Endothelial Cell Relaxants.- II. Lipoproteins and Proteins of the Arterial Wall.- Uptake of Intact Plasma Lipoproteins into the Arterial Wall of the Rabbit.- Beta-Lipoprotein Entry into the Arterial Wall and its Prevention.- Identification of Lipoproteins Involved in Human Atherosclerosis.- Quantitative Interrelationships Between Plasma Constituents and Normal and Atherosclerotic Human Intimai Tissue.- III. Lipids of the Arterial Wall.- Lipid Metabolic Pool in Subcellular Fractions of Rabbit and Human Atherosclerotic Lesions.- Glucose and Lipid Metabolism in the Human Arteriosclerotic Aorta.- Cholesterol Transfer in Vitro Between the Rabbit Aorta and Serum Lipoproteins.- Arterial Lipid Metabolism in Diabetic Animal Models with Reduced or Elevated Plasma Insulin Levels.- Lipid Metabolism in Perfused Human Coronary Arteries and Saphenous Veins.- IV. Enzymes of the Arterial Wall.- Workshop: Arterial Wall Enzymes.- A Comparison of the Rates of Lipolysis and Lipogenesis in the Rat Aorta.- Properties and Positional Specificity of Lipases in the Human Aorta.- The Influence of Atherosclerosis on Cholesterol Esterifying Activity of Rabbit and Monkey Aorta.- Aortic Uptake of Chylomicron Triglyceride in Vivo and Aortic Lipoprotein Triglyceride Lipase in Rat.- V. Metabolic Functions of the Arterial Cell.- Workshop: Metabolism and Function of Arterial Cells in Culture.- Cell Proliferation and Ultrastructural Changes in Regressing Atheroslerotic Lesions After Reduction of Serum Cholesterol.- Glucosamineglycans in Cell Membranes and the Extracellular Compartment of the Arterial Wall.- Increased Mitotic Activity in Primary Cultures of Aortic Medial Smooth muscle Cells After Exposure to Hyperlipemic Serum.- The Synthesis and Secretion of Sulfated Glycosaminoglycans by Vascular Smooth Muscle Cells In Vitro.- Etiology of Myointimal Cell Proliferation.- VI. Permeability of the Arterial Wall and Connective Tissue Reactions.- Workshop: Factors Affecting Permeability and Connective Tissue Reactions in Atherosclerosis.- Association of Coronary Artery Endothelial Injury with Elevation of Serum Bile Acids in the Rat.- Effect of Nicotine on Severity of Acute Myocardial Ischemic Injury.- Endothelial Permeability: Focal and Regional Patterns of 131I-Albumin and 131I-Fibrinogen Uptake and Transmural Distribution in the Pig Aorta.- Increased Collagen Synthesis in Early Rabbit Atherosclerosis and its Inhibition by Cis-Hydroxyproline.- Basement Membranes in Muscle Capillaries of Patients with Hyperlipidemias.- Effect of Adrenocortical Hormones on the Integrity of Rat Aortic Endothelium.- VII. Platelets-Thrombogenesis-Fibrinogen-Fibrinolysis.- Thrombosis and Atherosclerosis.- The Natural History of Experimentally Induced Mural Thrombi in Systemic Arteries of Normocholesterolaemic and Hypercholesterolaemic Monkeys.- Behavior of Fibrinogen and Fibrinolysis in Type II and Type IV Hyperlipemic Subjects and in Patients with Coronary Heart Disease.- Control of Platelet Function in Postmyocardial Infarction Patients During a Two Year Aspirin Study.- Dietary Fat, Platelet Function and Coronary Heart Disease in Man.- Enhanced Platelet Aggregation as a Risk Factor for Thromboembolic Complications in Atherosclerosis.- VIII. Animal Models of Atherosclerosis.- Workshop: Animal Models of Atherosclerosis.- Animal Models of Atherosclerosis and/or Myocardial Infarction.- Differences Between Human and Animal Atherosclerosis.- Experimental Atherosclerosis Due to Ascorbic Acid Deficiency.- On Pathogenetic Factors Concerned with Vascular Lesions Induced by High Sucrose Di